(1) Thyroid replacement led to resolution of both apnea and depression.
(2) We found that, compared to one- and two-dose infants, those treated with three doses of Exosurf were more premature, smaller, required a longer ventilator course, and had more frequent complications, including patent ductus arteriosus (PDA), intraventricular hemorrhage, nosocomial pneumonia, and apnea.
(3) The 14-fold increase in prolonged apnea frequency immediately following regurgitation supports the hypothesis for a causal relationship between apnea and regurgitation.
(4) When apnea is secondary to a disease process, the main focus should be on the identification of that disease process and on the institution of appropriate therapy.
(5) Particularly, the passive mechanism concept to explain obstructive sleep apnea during REM sleep advocated by Remmers and Guilleminault has substantially contributed to the recent development of research activities in this field.
(6) Three subjects breathed, two after less than 2 minutes of apnea, when PaCO2 was 47 and 54 mm Hg, and one after 4.5 minutes, when PaCO2 was 47 mm Hg.
(7) Apnea monitoring did not prevent, and in fact perpetrated the illusion of SIDS in this infant.
(8) The nine children who had a normal ZMD and improvement in their respiratory symptoms with the successful control of GER had advanced esophagitis (n = 4), central apnea (n = 3), or associated disorders in which respiratory symptoms improved with the avoidance of oral feedings (n = 2).
(9) It is suggested that the presence of abnormal OORR in sleep apnea may reflect a basic defect in pontomedullary control of respiration during sleep.
(10) Selecting therapy for a patient with obstructive sleep apnea requires a comprehensive evaluation including polysomnography, special examinations of the upper airway and assessing the cardiopulmonary status.
(11) The prevalence of sleep apnea, apnea index, duration of the longest episode of apnea, and penile rigidity were tabulated.
(12) The effect of alcohol on respiratory drive and on the apnea reflex response should be considered when humans ingest alcohol, in particular by those participating in water sports.
(13) These results are interpreted to signify that (1) inspiratory inhibitory inputs are more susceptible to depression by PB than inspiratory drive mechanisms; (2) the breathing pattern of apneusis results when summed inspiratory inhibition is reduced below a critical minimum level; (3) vagal and pneumotaxic center inhibitions on inspiration are equally weighted at apneusis, but not at apnea.
(14) The correlation of this EMG activity with SaO2 or the duration of obstructive apnea was more remarkable in submental EMG than dilator nasalis+ EMG in the observed patients.
(15) During hypocapnic apnea, some I, IE and E neurons became silent.
(16) To review the physiologic basis for normal and abnormal vagal reflexes arising from the pharynx, larynx, and esophagus, as well as the relevance of vagal reflexes to the pathogenesis of such clinically common cardiorespiratory responses as bradycardia, tachycardia, dysrhythmia, coronary angiospasm, bronchospasm, laryngospasm, prolonged apnea, and singultus (hiccups).
(17) A high incidence of sleep apnea (SA) and sleep-related periodic leg movements (PLMs) is also suspected.
(18) Continuous positive airway pressure (CPAP) is an accepted and highly effective method used in the treatment of obstructive sleep apnea (OSA).
(19) A rare case of idiopathic laryngeal spasm presented itself as sleep apnea in a middle-aged man.
(20) In COPD, the number of apneas was also correlated to the delta index (r2 = 0.92, p less than 0.01).
Apnoea
Definition:
(n.) Partial privation or suspension of breath; suffocation.
Example Sentences:
(1) Four patients developed apnoea after epidural administration of 50 micrograms of sufentanil.
(2) Diclofenac sodium suppositories 150-200 mg day-1 were compared with placebo in a double-blind study during the first 3 days after uvulopalatopharyngoplasty in 40 patients with habitual snoring or obstructive sleep apnoea syndrome.
(3) The syndrome of obstructive sleep apnoea is associated with an increased morbidity (the consequence of diurnal hypersomnolence and cardiovascular complications).
(4) By means of the adaptive estimation of the variance of respiratory movements, an amplitude-time window is calculated to choose between epochs with breaths and apnoea.
(5) In very low birthweight cases the risk factors identified were those associated with prolonged or recurrent hypoxia (recurrent apnoea, respiratory distress, assisted ventilation, and umbilical artery catheterisation).
(6) In patients with obstructive sleep apnoea the upper airway is prone to collapse during inspiration when the patient is supine, even when awake; this tendency can be reversed by breathing carbon dioxide.
(7) These effects were not accompanied by cardiac arrhythmias, with the exception of one animal, in which an unusually prolonged period of apnoea occurred.3.
(8) A patient with acromegaly was shown to have obstructive sleep apnoea by polygraphic recordings.
(9) Veratrine injected intravenously produced apnoea, bradycardia and long-lasting hypotension.
(10) In patients suspected of an obstructive sleep apnoea syndrome, however, it will always be necessary to perform further stage-wise diagnosis to safeguard the diagnosis.
(11) lignocaine, when plasma concentrations of lignocaine exceeded 4.7 micrograms ml-1, tracheal irritation elicited only brief apnoea.
(12) The combined effects of negative intrathoracic pressure swings during obstructive sleep apnoeas (OSAs) and increased sympathetic nervous system tone associated with hypoxia and sleep arousal may lead to pulmonary oedema or left-ventricular hypertrophy.
(13) The electrical activity of both expiratory and inspiratory muscles was inhibited during end-expiratory apnoea phase following injection of pdg into the right atrium.
(14) Stimulation of the nasal mucous membrane with ether vapour or water caused apnoea, bradycardia, a rise in arterial blood pressure and an increase in adrenal medullary catecholamine secretion.3.
(15) Seven male patients with obstructive sleep apnoea exhibited a paradoxical rise in both urine and sodium output during the hours of sleep.
(16) over 5 min) produced immediate disturbances in respiration (including dyspnoea and apnoea), blood pressure and heart rate followed by severe hypotension (mean systemic blood pressure less than 50 mmHg) or death due to respiratory and circulatory failure within 196 min.
(17) Although anatomical abnormalities of the upper airway have been recorded in some patients with obstructive sleep apnoea (OSA), a muscle tone dysregulation also seems to have an important role in this disorder.
(18) The variability between patients in the site(s) of upper airway obstruction during obstructive apnoeas have important implications for the choice of appropriate treatment in patients with obstructive sleep apnoea.
(19) The apparent excess risk is probably due to the consequences of sleep apnoea rather than snoring itself.
(20) In chemoreceptor-denervated animals, secondary depression led to central apnoea.