(n.) Sudden diminution or loss of consciousness, sensation, and voluntary motion, usually caused by pressure on the brain.
Example Sentences:
(1) There was no adrenal apoplexy or extensive haemorrhage that could explain shock in these patients.
(2) Acute symptomatic failure of the pituitary gland (pituitary apoplexy) commonly occurs in patients who have asymptomatic pituitary tumors.
(3) Catastrophic haemorrhage may occur even in small pituitary tumours and may result in the clinical syndrome of apoplexy with or without subsequent hypopituitarism.
(4) In 12 cases of pituitary apoplexy, a preexisting unsuspected adenoma was found.
(5) Therefore, our retrospective study suggests that pituitary apoplexy is not uncommon and has an acute clinical presentation.
(6) Pituitary apoplexy is an unusual manifestation of metastatic pituitary disease.
(7) In rats, a single administration of acrylonitrile (vinyl cyanide) produces a rapidly occurring bilateral adrenal apoplexy.
(8) Total and coronary mortality rates have been determined and also the incidence of certain non-fatal states which required hospitalization due to various types of coronary heart disease (CHD), hypertension, brain apoplexy, diabetes mellitus and malignancies.
(9) Histologically an adrenal apoplexy with necrosis and in liver, spleen and lung a lot of neutrophils were seen.
(10) They illustrate the difficulty of differentiating pituitary apoplexy from other acute neurologic conditions.
(11) In the majority of apoplexy patients the absence of a primary haemorrhage points to acute vascular occclusion with regional ischemia as the initiating event.
(12) Pituitary apoplexy is a syndrome with variable clinical manifestations depending on which parasellar structures (such as the optic nerves and chiasm, cavernous and sphenoid sinuses, or the hypothalamus) are compressed when the pituitary undergoes rapid enlargement.
(13) Pituitary apoplexy, a rare but life-threatening condition, may be highly variable in its clinical appearance and therefore should be considered in any patient with abrupt neurologic deterioration.
(14) Postpartum abdominal apoplexy is a rare obstetric complication that is associated with a very high maternal mortality rate.
(15) During the first year after the pituitary apoplexy, severe proliferative retinopathy developed in the left eye, which became almost blind.
(16) This report deals with a detailed course of one patient with acromegaly who had a pituitary apoplexy.
(17) A case of acromegaly complicated by pituitary apoplexy is described.
(18) The aim of surgical intervention is primarily to prevent ischemia and simultaneous cerebral apoplexy, and only after this to prevent the progressions of the existing ischemic changes.
(19) Pituitary apoplexy is characterized by a wide spectrum of clinical features.
(20) A patient, 38-year-old man, with hemorrhage into a prolactin-secreting pituitary adenoma, or pituitary apoplexy, is reported.
Brain
Definition:
(n.) The whitish mass of soft matter (the center of the nervous system, and the seat of consciousness and volition) which is inclosed in the cartilaginous or bony cranium of vertebrate animals. It is simply the anterior termination of the spinal cord, and is developed from three embryonic vesicles, whose cavities are connected with the central canal of the cord; the cavities of the vesicles become the central cavities, or ventricles, and the walls thicken unequally and become the three segments, the fore-, mid-, and hind-brain.
(n.) The anterior or cephalic ganglion in insects and other invertebrates.
(n.) The organ or seat of intellect; hence, the understanding.
(n.) The affections; fancy; imagination.
(v. t.) To dash out the brains of; to kill by beating out the brains. Hence, Fig.: To destroy; to put an end to; to defeat.
(v. t.) To conceive; to understand.
Example Sentences:
(1) Here we have asked whether protection from blood-borne antigens afforded by the blood-brain barrier is related to the lack of MHC expression.
(2) The extents of phospholipid hydrolysis were relatively low in brain homogenates, synaptic plasma membranes and heart ventricular muscle.
(3) First results let us assume that clinically silent TIAs also (in analogy to clinically silent brain infarctions) could be detected and located.
(4) Brain and ganglia of embryonic Periplaneta americana were grown for 2 to 3 weeks in a chemically defined medium.
(5) The results indicated that neuropsychological measures may serve to broaden the concept of intelligence and that a brain-related criterion may contribute to a fuller understanding of its nature.
(6) To quantify the size of the lesion in mice, the area of the infarct on the brain surface was assessed planimetrically 48 h after MCA occlusion by transcardial perfusion of carbon black.
(7) However, CT will be insensitive in the detection of the more cephalic proximal lesions, especially those in the brain stem, basal cisterns, and skull base.
(8) The fine structure of neurofibrillary tangles in the hippocampal gyrus, substantia nigra, pontine nuclei and locus coeruleus of the brain was postmortem studied in a case of progressive supranuclear palsy.
(9) We similarly evaluated the ability of other phospholipids to form stable foam at various concentrations and ethanol volume fractions and found: bovine brain sphingomyelin greater than dipalmitoyl 3-sn-phosphatidylcholine greater than egg sphingomyelin greater than egg lecithin greater than phosphatidylglycerol.
(10) This study examined the [3H]5-HT-releasing properties of 3,4-methylenedioxymethamphetamine (MDMA) and related agents, all of which cause significant release of [3H]5-HT from rat brain synaptosomes.
(11) In the present investigation we monitored the incorporation of [14C] from [U-14C]glucose into various rat brain glycolytic intermediates of conscious and pentobarbital-anesthetized animals.
(12) Cranial MRI revealed delayed myelination in the white matter but no brain malformation.
(13) When compared with lissencephalic species, a great horizontal fibrillary system (which is vertically arranged in gyral regions) was observed in convoluted brains.
(14) In more than 70 per cent of these, brain injury is the decisive lethal factor.
(15) Brain damage may be followed by a number of dynamic events including reactive synaptogenesis, rerouting of axons to unusual locations and altered axon retraction processes.
(16) Based on our results, we propose the following hypotheses for the neurochemical mechanisms of motion sickness: (1) the histaminergic neuron system is involved in the signs and symptoms of motion sickness, including vomiting; (2) the acetylcholinergic neuron system is involved in the processes of habituation to motion sickness, including neural store mechanisms; and (3) the catecholaminergic neuron system in the brain stem is not related to the development of motion sickness.
(17) The regional distribution of beta-adrenoceptor subtypes was found to be similar to that seen in the rat brain.
(18) We assessed changes in brain water content, as reflected by changes in tissue density, during the early recirculation period following severe forebrain ischemia.
(19) If tracer is introduced into the carotid artery after osmotic treatment, brain uptake is increased by a net factor of 50 (a factor of 70 due to elevation of PA, multiplied by 7 due to infusion by the carotid route) as compared to uptake by normal, untreated brain with infusion into a peripheral vein.
(20) This study examined both the effect of variations in optical fiber tip and in light wavelength on laser-induced hyperthermia in rat brain.