What's the difference between bifacial and surface?

Bifacial


Definition:

  • (a.) Having the opposite surfaces alike.

Example Sentences:

  • (1) "In terms of the Out of Africa event, new dating of the Dmanisi site in Georgia places some of the material from there older than 1.8m years ago, so it is evident that human emergence from Africa preceded even this new date for bifacial tools.
  • (2) The onset of the Melkersson-Rosenthal syndrome, manifested by bifacial paralysis and severe edema of the upper lip in a 25-year-old man, was followed shortly by signs of secondary syphilis.
  • (3) Populations could have experimented with bifacial working many times before it took hold more widely around 1.6m years ago."
  • (4) Five patients had bifacial microsomia, and 44 patients had hemifacial microsomia.
  • (5) In the Fe-deficient late exponential cells there are long, bifacial pieces of RER (one side rough and the other smooth) which later undergo degradation.
  • (6) We report a case of unilateral transient and reversible facial paresis, which was superimposed on a congenital bifacial palsy in a young adult with Moebius syndrome.

Surface


Definition:

  • (n.) The exterior part of anything that has length and breadth; one of the limits that bound a solid, esp. the upper face; superficies; the outside; as, the surface of the earth; the surface of a diamond; the surface of the body.
  • (n.) Hence, outward or external appearance.
  • (n.) A magnitude that has length and breadth without thickness; superficies; as, a plane surface; a spherical surface.
  • (n.) That part of the side which is terminated by the flank prolonged, and the angle of the nearest bastion.
  • (v. t.) To give a surface to; especially, to cause to have a smooth or plain surface; to make smooth or plain.
  • (v. t.) To work over the surface or soil of, as ground, in hunting for gold.

Example Sentences:

  • (1) The resulting dose distribution is displayed using traditional 2-dimensional displays or as an isodose surface composited with underlying anatomy and the target volume.
  • (2) Phospholipid methylation in human EGMs is distinctly different from that in rat EGMs (Hirata and Axelrod 1980) in that the human activity is not Mg++-dependent, and apparent methyltransferase I activity is located in the external membrane surface.
  • (3) To quantify the size of the lesion in mice, the area of the infarct on the brain surface was assessed planimetrically 48 h after MCA occlusion by transcardial perfusion of carbon black.
  • (4) Using monoclonal antibodies directed against the plasma membrane of Madin-Darby canine kidney (MDCK) cells, we demonstrated previously that a glycoprotein with an Mr = 23,000 (gp23) had a non-polarized cell surface distribution and was observed on both the apical and basolateral membranes (Ojakian, G. K., Romain, R. E., and Herz, R. E. (1987) Am.
  • (5) In the surface epithelial cells, the basolateral cell surface showed moderate enzymatic activity.
  • (6) Such an increase in antibody binding occurred simultaneously with an increase in the fluidity of surface lipid regions, as monitored by fluorescence depolarization of 1-(trimethylammoniophenyl)-6-phenyl-1,3,5-hexatriene.
  • (7) The role of Ca2+ in cell agglutination may be either to activate the cell-surface dextran receptor or to form specific intercellular Ca2+ bridges.
  • (8) The subcellular distribution of sialyltransferase and its product of action, sialic acid, was investigated in the undifferentiated cells of the rat intestinal crypts and compared with the pattern observed in the differentiated cells present in the surface epithelium.
  • (9) Even with hepatic lipase, phospholipid hydrolysis could not deplete VLDL and IDL of sufficient phospholipid molecules to account for the loss of surface phospholipid that accompanies triacylglycerol hydrolysis and decreasing core volume as LDL is formed (or for conversion of HDL2 to HDL3).
  • (10) A total of 555 caries lesions were registered on proximal surfaces, 49.1% being primary lesions in the enamel, 21.4% primary lesions into the dentin and 29.5% secondary lesions.
  • (11) Contact angles of Silafocon A and PMMA were relatively uninfluenced by front surface radii between 7.7 and 8.85 and 7.3 to 8.8 mm, respectively.
  • (12) These cells contained organelles characteristic of the maturation stage ameloblast and often extended to the enamel surface, suggesting a possible origin from the ameloblast layer.
  • (13) Together these observations suggest that cytotactin is an endogenous cell surface modulatory protein and provide a possible mechanism whereby cytotactin may contribute to pattern formation during development, regeneration, tumorigenesis, and wound healing.
  • (14) Our Ph1-positive ALL revealed B-cell lineage leukemia, since their surface phenotype were Ia+ and CD10+ and they have rearranged immunoglobulin JH genes.
  • (15) The complete nucleotide sequence of the gene for a cell surface protein antigen (SpaA) of Streptococcus sobrinus MT3791 (serotype g) was determined.
  • (16) To investigate the mechanism of enhanced responsiveness of cholesterol-enriched human platelets, we compared stimulation by surface-membrane-receptor (thrombin) and post-receptor (AlF4-) G-protein-directed pathways.
  • (17) Lysis of EAC4b,3b cellular intermediates formed to contain a low surface amount of C3b was more inhibited than was lysis of cells formed with a standard amount of C3b on the surface.
  • (18) After either 5 or 10 days of culture with both cytokines, intense immunofluorescent staining for Ia could be identified on the surface of greater than 80-90% of the viable islet cells.
  • (19) Within the capillary-perfused mucosa and muscularis (between 50 and 2000 microns from the urothelial surface), concentrations decreased by 50% for each 500-microns distance.
  • (20) Displacement of the surface of the cornea of bovine eyes after disruption of intact structures was investigated by means of holographic interferometry.

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