(1) No age differences were observed in DBH activity of any tissues examined or in the activities of any enzymes in the coeliac-mesenteric ganglion complex.
(2) This preliminary study shows an adrenergic control system composed of chromaffin cells and adrenergic nerves similar to that found in other teleosts investigated, although the systemic arteries (coeliac artery, dorsal aorta and the vasculature of the air-breathing organ) appear to lack an adrenergic innervation.
(3) A 17-year-old boy who had been treated for insulin-dependent diabetes since age 2, and for coeliac disease since age 6, presented a major growth retardation (-6 SD), a delayed puberty and a hepatomegaly with excessive glycogen storage (Mauriac's syndrome).
(4) Four patients with coeliac disease, who had shown complete mucosal recovery after prolonged treatment with a strict gluten-free diet, volunteered to consume oats in addition to their gluten-free diet for a period of one month and were studied by jejunal biopsy before and after the experimental period.
(5) Serum levels of dialysed triiodothyronine and thyroxine and of triiodothyronine are significantly lower in coeliac patients than in normal infants (P less than 0.01).
(6) Literature studies and the results of the present study indicate that CT-guided or ultrasound-guided techniques should be preferred in coeliac plexus block whenever the facilities are available.
(7) It has been shown that GGT activity in the duodenal biopsy homogenates of the children with coeliac disease (n-10) in remission (1 to 3 years of gluten-free diet) is lower than in those with other gastrointestinal tract diseases (n-6).
(8) This study finally confirms that it is impossible to use organotypic cultures as an in vitro test for the diagnosis of coeliac disease.
(9) Coeliac disease is a primary malabsorption syndrome, whose gastrointestinal symptomatology regresses following a gluten-free diet.
(10) In this patient, azathioprine induced and sustained a remission when unacceptably high doses of prednisone had failed, and may prove to be a valuable immunosuppressive in non-responsive coeliac disease.
(11) Histological examination of biopsy specimens revealed jejunal mucosal villous atrophy compatible with coeliac disease in 13 of 122 relatives.
(12) In remission he developed coeliac disease, controlled by dietary measures, but 26 months after the end of chemotherapy a severe dyspeptic syndrome appeared; endoscopy and barium meal suggested the presence of a gastric tumour, which was surgically removed and showed the histological features of a non-Hodgkin's lymphoma, lympho-histiocytic type.
(13) Stenosis and occlusion of the coeliac trunk and the superior mesenteric artery can be detected.
(14) Both patients were cured by surgical decompression of the coeliac trunk.
(15) Subclinical coeliac disease appears to be unusually over-represented among patients with idiopathic osteoporosis, and screening for gliadin antibodies might therefore be a valuable addition to the routine assessment of the osteopenic patient.
(16) Though the levels in Crohn's disease were significantly higher than those in ulcerative colitis and in coeliac disease, there was marked overlap between the disorders and control subjects, and so they were of no value in differential diagnosis.
(17) The authors believe that coeliac disease, as defined at present, is a heterogeneous condition.
(18) The splanchnic fibers originate from the single coeliac ganglion, situated near the third spinal nerve on the right side, and pass caudally to the islet.
(19) In untreated coeliac patients the distribution of the various cell types was essentially unchanged, although the number of these cells was markedly increased, including those which expressed the Ta1 antigen.
(20) 5- and 24-hour urinary excretion rates of xylose were of no value in discriminating between patients with and without coeliac disease; similarly, the 3-hour blood xylose concentration was nondiscriminatory.
Gluten
Definition:
(n.) The viscid, tenacious substance which gives adhesiveness to dough.
Example Sentences:
(1) In October, an episode of South Park saw the whole town go gluten-free (the stuff, it was discovered, made one’s penis fly off).
(2) The gluten-free diet failed to improve the neurologic disability except in 1 patient.
(3) Four patients with coeliac disease, who had shown complete mucosal recovery after prolonged treatment with a strict gluten-free diet, volunteered to consume oats in addition to their gluten-free diet for a period of one month and were studied by jejunal biopsy before and after the experimental period.
(4) Onset of disease was positively correlated to the duration of breast-feeding and not related to the age at gluten introduction.
(5) Neither the non-restricted diet nor the gluten-free diet had any effect on gastric morphology, the ability to secrete gastric acid, serum gastrin levels, or the frequency or titres of circulating parietal cell antibodies.
(6) A report by Mintel found 15% of households were avoiding gluten and wheat – more than half because they believed it was part of a healthy diet.
(7) In 2011, a study of people with irritable bowel syndrome found that subjects felt better when they ate a gluten-free diet .
(8) It has been shown that GGT activity in the duodenal biopsy homogenates of the children with coeliac disease (n-10) in remission (1 to 3 years of gluten-free diet) is lower than in those with other gastrointestinal tract diseases (n-6).
(9) We conclude that 1) there is a significant improvement in clinical disease activity on an elemental diet, independent of gluten administration, 2) small bowel morphology improves rapidly on an elemental diet, and 3) complement deposition but neither IgA deposition nor circulating antibody levels correlate with gluten intake.
(10) Three children had normal mucosal appearance after 58 to 73 months on normal diets, one of whom showed temporary mucosal abnormalities, another having occasionally low enzymes, in both suggesting underlying gluten sensitivity.
(11) The presence of anti-GL pIgA in serum seems related to chronic exposure to gluten of CD patients with a flat intestinal mucosa.
(12) The absence of rejection in gluten-fed rats may be explained by an impaired cellular immune response due to protein malnutrition.
(13) Coeliac disease is a primary malabsorption syndrome, whose gastrointestinal symptomatology regresses following a gluten-free diet.
(14) Features suggestive of a latent gluten-sensitive enteropathy were found in one of the other six DH patients; he developed disaccharidase deficiencies and villus atrophy when 20 g gluten was added to his usual gluten-containing diet.
(15) In contrast, none of 16 patients with enteropathy associated T cell lymphoma had raised levels of alpha gliadin antibody, and treatment with a gluten free diet resulted in histological improvement in one and transient clinical improvement in six patients.
(16) Determination of plasma enteroglucagon may facilitate the control of the dietary adherence during gluten-free diet and may in some children indicate mucosal relapse during gluten challenge.
(17) The changes in the quantity of protein and energy consumed by the rats, which were brought about by the addition of the essential limiting amino acids and groups of essential amino acids to gluten, casein, and zein, were not linearly related to alterations in the total plasma amino acid concentrations or to the accumulation of the added amino acids in the blood plasma.
(18) Wet corn gluten feed was fed at 0 (control), 10, 20, and 30% of the total ration DM.
(19) We concluded that 82.4% of patients had hyposplenic changes and that there was a non significant tendency to improve such changes by a strict adherence to gluten-free diet.
(20) N balance was negative during the preliminary period when no infusion was given; infusion of casein or gluten increased the daily N retention to 5-15 g. 3.