(1) To investigate the relationship between Helicobacter pylori infection and nonsteroidal anti-inflammatory drug (NSAID) intolerance and the effect of gold use on the seroprevalence of H. pylori.
(2) The authors conclude that H. pylori alone causes little or no effect on an intact gastric mucosa in the rat, that either intact organisms or bacteria-free filtrates cause similar prolongation and delayed healing of pre-existing ulcers with active chronic inflammation, and that the presence of predisposing factors leading to disruption of gastric mucosal integrity may be required for the H. pylori enhancement of inflammation and tissue damage in the stomach.
(3) A remarkably close relationship was found between both H. pylori urease subunits and jack bean (Canavalia ensiformis) urease, the subunit of which is a single 840 amino acid polypeptide.
(4) The pathomechanism, how C. pylori facilitates the development of peptic ulcer is since hypothetical.
(5) Sixty of 65 H. pylori-infected adults were correctly identified by the test.
(6) The Radio-PAGE and immunoblot typing methods both gave precise identification of Helicobacter pylori strains, but Radio-PAGE was found to give higher resolution and represents a standardised universally applicable fingerprinting method for Helicobacter pylori.
(7) The high rate of H pylori ammonia production in uraemic patients should accentuate any ammonia induced effects.
(8) Almost all the transplant patients with H. pylori antibodies were taking steroids (98%) compared to 84% of those without antibodies (p less than 0.05).
(9) Helicobacter pylori infection is virtually always present in duodenal and gastric ulcer and active chronic gastritis, but not prepyloric ulcer.
(10) Considerable knowledge has recently accumulated on the mechanism by which Helicobacter pylori (H. pylori) induces chronic gastritis.
(11) Previous use of metronidazole was reported in only 16 patients, 11 of whom (68.8%) harboured resistant Helicobacter pylori strains.
(12) Helicobacter pylori has been demonstrated as an etiologic agent of human gastritis and peptic ulcer formation.
(13) C. pylori was cultivated from the biopsy specimens of 41 of these patients, and 31 were culture-negative.
(14) These findings suggest that H pylori may add to the local production of paf in inflamed gastric mucosa.
(15) Since pathways inhibiting gastric secretion are defective in patients with duodenal ulcers, we investigated whether H pylori interferes with the normal gastric inhibition that is mediated by somatostatin.
(16) Specimens obtained at gastric biopsies performed for suspected acid peptic disease in patients 5 through 17 years of age were retrospectively reviewed for the presence of Campylobacter pylori (CP), a gram-negative bacillus associated with chronic gastritis and peptic ulcer disease in adults.
(17) All five rhesus monkeys that received the strain isolated from rhesus monkeys became H. pylori positive by day 14 and remained positive through day 56 Antral inflammation developed in all monkeys.
(18) Daily administration of suspensions of H. pylori organisms to sham-operated rats with intact gastric mucosa, however, resulted in no ulceration or inflammation despite identification of surface H. pylori organisms at death.
(19) Active, chronic gastritis is characteristic of Campylobacter pylori colonisation.
(20) Three volunteers infected with C. pylori had the highest IgG antibody titers of the group.
Pyloric
Definition:
(a.) Of, pertaining to, or in the region of, the pylorus; as, the pyloric end of the stomach.
Example Sentences:
(1) The vagally induced pyloric contraction was resistant to atropine but sensitive to SPA and hexamethonium, indicating involvement of SP in the activation of preganglionic neurons as well.
(2) Gastric juice was examined in terms not only of conventional indices, observed volume, titratable acidity and acid output, but also Vg, the volume corrected for pyloric loss and duodenal reflux.
(3) No evidence was found of sex ratio anomalies in other classes of relative of pyloric stenosis index cases, whether or not the index cases had affected sibs.
(4) Pyloric sphincter pressure was assessed with water-perfused polyvinyl tubes.
(5) Nineteen patients presented as emergencies with haemorrhage, perforation, pyloric stenosis or intestinal obstruction.
(6) Antral mucosal diaphragm is uncommon, and presents with manifestations of obstruction to the pyloric outlet.
(7) MAb FW6 was also reactive with pyloric mucus glands, Brunner's glands of the duodenum, Paneth cells of the ileum, pancreatic ducts, absorptive cells of the right colon, bronchiolar glands, kidney urothelia, and with a restricted number of normal mucinous tubuli of salivary gland.
(8) Gastrin I did not significantly increase pyloric pressure but did antagonize the pyloric response to duodenal acidification.
(9) The term gastroduodenal fistula is suggested to describe double pyloric canal.
(10) Report of a case of perforation of a gallstone into the duodenal bulb producing few signs but the clinical picture of pyloric stenosis.
(11) L-NAME enhanced pyloric contractions to intra-arterial acetylcholine.
(12) The fasting and postprandial serum gastrin levels were high in one patient with recurrence, whose antrum was preserved as long as 3 cm proximal to the pyloric ring; this was longer than that described in our original method of PPG.
(13) In a search of features that might be relevant to the understanding of the hyperbilirubinaemia of infants with hypertrophic pyloric stenosis (HPS), we examined the duodenal fluid in 11 infants with this condition.
(14) A similar relationship is observed between the pyloric sphincter and duodenal motility.
(15) Tissue alterations included mechanical compression of the pancreas and liver, granulomatous inflammation and necrosis of the liver, and trauma to the muscularis externa of the pyloric caeca.
(16) The present study reconciles this conflict by showing that the major form of gastrin in the pyloric antrum is the heptadecpeptide form, while the duodenum contains mainly "big" and almost no heptadecapeptide gastrin.
(17) From 1950 to 1984, 679 patients were treated for infantile hypertrophic pyloric stenosis (IHPS) in the County of Funen, Denmark.
(18) This communication describes the removal of such a pedunculated distal antral polyp which not only caused intermittent pyloric obstruction but also a moderately severe iron deficiency anemia in an 82-year old female.
(19) The glandular stomach carcinogens N-methyl-N'-nitro-N-nitrosoguanidine, N-ethyl-N'-nitro-N-nitrosoguanidine, N-propyl-N'-nitro-N-nitrosoguanidine, 4-nitroquinoline 1-oxide and N-nitroso-N-methylurethane induced up to 100-fold increase in ODC activity in the pyloric mucosa of the stomach; the activity was maximal 24 hr after administration and returned to the control level after 48 to 72 hr.
(20) Pyloric stenosis and retention point to the failure of the operation via the negatively effected subjective feeling of well-being.