(v. t.) To lease again; to grant a new lease of; to let back.
(n.) To let loose again; to set free from restraint, confinement, or servitude; to give liberty to, or to set at liberty; to let go.
(n.) To relieve from something that confines, burdens, or oppresses, as from pain, trouble, obligation, penalty.
(n.) To let go, as a legal claim; to discharge or relinquish a right to, as lands or tenements, by conveying to another who has some right or estate in possession, as when the person in remainder releases his right to the tenant in possession; to quit.
(n.) To loosen; to relax; to remove the obligation of; as, to release an ordinance.
(n.) The act of letting loose or freeing, or the state of being let loose or freed; liberation or discharge from restraint of any kind, as from confinement or bondage.
(n.) Relief from care, pain, or any burden.
(n.) Discharge from obligation or responsibility, as from debt, penalty, or claim of any kind; acquittance.
(n.) A giving up or relinquishment of some right or claim; a conveyance of a man's right in lands or tenements to another who has some estate in possession; a quitclaim.
(n.) The act of opening the exhaust port to allow the steam to escape.
Example Sentences:
(1) The fluoride treated specimens released more fluoride than the nontreated ones.
(2) In conclusion, in S-rats a glucose-stimulated insulin release is accompanied by an increase in IBF, but this is not observed in P-rats.
(3) These are typically runaway processes in which global temperature rises lead to further releases of CO², which in turn brings about more global warming.
(4) This study examined the [3H]5-HT-releasing properties of 3,4-methylenedioxymethamphetamine (MDMA) and related agents, all of which cause significant release of [3H]5-HT from rat brain synaptosomes.
(5) The vascular endothelium is capable of regulating tissue perfusion by the release of endothelium-derived relaxing factor to modulate vasomotor tone of the resistance vasculature.
(6) gamma-Aminobutyric acid (GABA) and glutamate release from the treated side was higher than the control value during the first 2-3 h, a result indicating an important role of glial cells in the inactivation of released transmitter.
(7) In contrast, the effects of deltamethrin and cypermethrin promote transmitter release by a Na+ dependent process.
(8) However, direct measurements of mediator release should be carried out to reach a firm conclusion.
(9) In addition to their involvement in thrombosis, activated platelets release growth factors, most notably a platelet-derived growth factor (PDGF) which may be the principal mediator of smooth muscle cell migration from the media into the intima and of smooth muscle cell proliferation in the intima as well as of vasoconstriction.
(10) During recovery glucose uptake was reduced and citrate release was unaffected.
(11) Bradykinin also stimulated arachidonic acid release in decidual fibroblasts, an effect which was potentiated in the presence of epidermal growth factor (EGF), but which was not accompanied by an increase in PGF2 alpha production.
(12) As prolongation of the action potential by TEA facilitates preferentially the hormone release evoked by low (ineffective) frequencies, it is suggested that a frequency-dependent broadening of action potentials which reportedly occurs on neurosecretory neurones may play an important role in the frequency-dependent facilitation of hormone release from the rat neurohypophysis.
(13) Release of 51Cr was apparently a function of immune thymus-derived lymphocytes (T cells) because it was abrogated by prior incubation of spleen cells with anti-thymus antiserum and complement but was undiminished by passage of spleen cells through nylon-wool columns.
(14) It is suggested that the normal cyclical release of LH is inhibited in PCO disease by a negative feedback by androgens to the hypothalamus or the pituitary, and that wedge resection should be reserved for patients in whom other forms of treatment have failed.
(15) Results suggest that Cd-MT is reabsorbed and broken down by kidney tubule cells in a physiological manner with possible subsequent release of the toxic cadmium ion.
(16) The latter result indicates that the dexamethasone block is upstream from release of esterified arachidonic acid.
(17) Treatment of the bound F1-ATPase with 4-chloro-7-nitrobenzofurazan prevented complete release of the enzyme by ATP.
(18) Glucose release from these samples was highly correlated with starch gelatinization (r2 = .99).
(19) Furthermore, H-7 enhanced the effect of thrombin on AA release.
(20) Because it has been suggested that the lathyrogen, BAPN, may stimulate the release of proteases, the protease inhibitors Trasylol and epsilon-aminocaproic acid (EACA) were given alone or in combination to BAPN-treated rats.