What's the difference between steapsin and triglyceride?
Steapsin
Definition:
(n.) An unorganized ferment or enzyme present in pancreatic juice. It decomposes neutral fats into glycerin and fatty acids.
Example Sentences:
(1) The methods, which involved digestion of membrane with Steapsin, a crude pancreatic extract containing diastase and trypsin, gel filtration and preparative electrophoresis on polyacrylamide, provided an enzyme with a high specific activity in good yield.
(2) Aggregates were formed when clear supernatants from hepatic microsomes that had been treated with steapsin were desalted and concentrated.
(3) The enzyme lacks NADPH dehydrogenase activity and is insensitive to treatment with 2-bromo-4'-nitroacetophenone and steapsin: it catalyses N-oxidation of imipramine, trimethylamine and NN-dimethylaniline in molar proportions considerably different from those of the cytochrome P-450-supported reactions.
(4) Comparisons are made among detergent-solubilized and protease (steapsin)-solubilized preparations of porcine liver reductases, showing unequivocally that the 31P NMR signals at approximately 0.0 ppm in the detergent-solubilized, hydrophobic form are attributable to phospholipids.
(5) The solubilized microsomal fraction treated with steapsin lost the reductase activity for hexadecanal but not that for cytochrome c. These results suggest that the aldehyde reductase in brain microsomes is identical to NADPH-cytochrome c reductase and that a hydrophobic portion of the NADPH-cytochrome c reductase is required for the reduction of hexadecanal.
(6) NAD+ glycohydrolase (NAD+ nucleosidase, EC 3.2.2.6) can be solubilized from calf spleen microsomes (microsomal fractions) by steapsin or by detergents to yield respectively a hydrophilic (i.e.
Triglyceride
Definition:
(n.) A glyceride formed by the replacement of three hydrogen atoms in glycerin by acid radicals.
Example Sentences:
(1) Their effects on various lipid fractions, viz., triglycerides (TG), phospholipids, free cholesterol, and esterified cholesterol, were studied in liver, plasma, gonads, and muscle.
(2) Statistically significant increases of triglycerides occurred under the combined preparations, of phospholipids under Ovosiston and Deposiston and of the beta-lipoproteins under Ovosiston and Gravistat.
(3) No significant difference was found for triglycerides, uric acid, albumin and gamma-globulins.
(4) There was no difference in triglyceride content or phospholipid species between WKY rats and untreated SHR, except for a higher cholesterol content in SHR.
(5) Triglyceride (Trigly) in female dogs, glutamic-pyruvic transaminase (GPT) and urea nitrogen (Urea-N) in male dogs tended to increase.
(6) AIS treatment lowered serum insulin levels, serum triglyceride levels, caloric intake, weight gain, liver weight, acetate 14C incorporation into cholesterol 14C, and the percentage of liver lipid that was cholesterol.
(7) Further significantly positive associations to the incidence of myocardial infarction (MI) were found for the following parameters: Systolic and diastolic blood pressure, family history of premature MI, cigarette smoking, plasma levels of triglycerides, VLDL-cholesterol and blood glucose.
(8) The same alleles were weakly associated with elevated levels of serum levels of cholesterol, triglycerides and apolipoprotein B.
(9) In fasted cats no changes were observed in plasma triglycerides or phospholipids.
(10) In comparison with native counterparts, the in vitro-formed LDL2 and HDL + VHDL were characterized by lower levels of triglyceride and cholesterol ester and higher levels of free cholesterol and lipid phosphorus.
(11) The triglycerides are isolated by means of thin-layer chromatography.
(12) Alternatively, the data presented herein strongly suggest that diets containing conventional quantities of fat, in which saturated fat is replaced by unsaturated fat and dietary cholesterol reduced, would result in the desired reductions to total and low-density lipoprotein cholesterol concentrations without the adverse effects of increased postprandial glucose and insulin concentrations, increased fasting and postprandial total and very-low-density lipoprotein triglyceride concentrations, and decreased fasting high-density lipoprotein cholesterol concentrations.
(13) There are statistically significant correlations between plasma triglycerides and predictors of fatness, particularly in the body, both in men and women.
(14) Decreases in the level of triglycerides and prebetalipoproteins were noted after filtering but the differences were not significant.
(15) The examination of the elution pattern of the triglyceride and cholesterol revealed that this large LDL was composed of a large amount of triglyceride.
(16) Insulin response was not correlated with KG, whereas it was significantly associated with higher triglyceride levels.
(17) Patients under caloric restriction showed a reduction in their total triglyceride content, a reduction in their content of unsaturated fatty acyl groups, and a relative increase in phospholipid content.
(18) In vivo hepatic rates of fatty acid and cholesterol synthesis determined in meal-fed normolipidemic rats were suppressed significantly by the oral administration of (--)-hydroxycitrate for 6 hr, when control animals exhibited maximal rates of lipid synthesis; serum triglyceride and cholesterol levels were significantly reduced by (--)-hydroxycitrate.
(19) The medium-chain triglyceride oil supplementation did not influence the growth of these infants.
(20) These variations in apo C-II were closely correlated with the plasma clearance of triglyceride.